Medical treatment of autism spectrum disorder in children: Current evidence, controversies, and clinical challenges Evidence of oxidative damage and inflammation associated with low glutathione redox status in the autism brain Translational Psychiatry Mitochondrial dysfunction reveals H2S mediated synaptic sulfhydration as a potential mechanism for autism associated social defects: Cell Metabolism Metabolic biomarkers of increased oxidative stress and impaired methylation capacity in children with autism The American Journal of Clinical Nutrition
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does low glutathione cause autism The impact of glutathione metabolism in autism spectrum disorder